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In preclinical models of schizophrenia, treatments with antioxidant agents, such as NAC, that increase glutathione levels were associated with improvement in cognitive and social behaviors [153,154,155], an elevation in the number of parvalbumin cells [156,157], prevention of deficits in evoked-related potentials measured by electroencephalography (EEG) electrodes and in the prepulse inhibition of the acoustic startle response [157], an increase in mitochondria numbers and myelin-related mRNA expression [155], with higher benefits if the administration occurred at an early stage of neurodevelopment
